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Extra resources for Advances in Electrocardiology 2004: Proceedings of the 31th International Congress on Electrocardiology (Advances in Electrocardiography)

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Although this channel is not down regulated in long lasting atrial fibrillation, it is not important for repolarization in the remodeled atria. In stead, channels active early during the plateau phase of the action potential (like Ito and Ikur) are more important and blocking these channels may effectively induce prolongation of the action potential. This has been investigated by Allessie and co-workers who showed that an Ikur blocker AVEO118 can cardiovert persistent atrial fibrillation by prolonging especially the very short action potentials of the remodeled atria.

Tamaki, T. Yamazaki and S. Nishimura Measurement of Intracardiac Bioimpedance in Rate Adaptive Pacemakers 737 738 A . Kuusik, R. Land, M. Min, T. Pane and G. Poola Ventricular Pacing Thresholds Following High-Energy Ventricular Defibrillation Shocks Y. Yamanouchi, S. Kodama, T. Ohta, N. Morito, E. Yahiro, K. Miyoshi and H. Urata Prevention of Atrial Fibrillation by Biatrial Pacing: The Outcome and the Electrophysiological Mechanism of Prevention Y. Enjoji, T. Noro, T. Nakae, N. Tezuka, K. Kumagai, T.

13 24. Priori SG, Schwartz PJ, Napolitano C, et al: Risk stratification in the long-QT syndrome. N Engl JMed 2003;348:1866-1874. 25. Splawski I, Shen J, Timothy KW, et al: Spectrum of mutations in long-QT syndrome genes. KVLQT1, HERG, SCNSA, KCNE1, and KCNE2. Circulation 2000;102:1178-1185. 26. Moss AJ, Zareba W, Kaufman ES, et al: Increased risk of arrhythmic events in long-QT syndrome with mutations in the pore region of the human ether-a-go-go-related gene potassium channel. Circulation 2002; 105:794-799.

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